So why was specific nerve pain in my mouth eliminated by the removal of my wisdom tooth the first time, and why was that same pain eliminated by an antiviral and nerve protecting protocol the second time around? I’m thinking it could be either…
A: The cause of the pain was, in fact, different in each case – the first time it stemmed from a decayed wisdom tooth, and the second time it was viral inflammation. And it just subjectively felt identical to me.
Or…
B: The wisdom tooth was never responsible for the pain at all – It got blamed the first time for what was really a viral injection of the nerve(s) in both cases. And it was the Nitrous Oxide, (laughing gas) I was given for the surgery that affected the nerve(s) (most pleasingly in this case!) beyond just the desired temporary anesthetic and analgesic effect.
My own bias is for B, because the pain felt identical to me in both instances; same sensation of pain, same location, and the antiviral protocol was effective and worked quickly.
Thoughts about B, and the effects of Nitrous Oxide:
In addition to being nerve calming and limiting the pain-signaling effect, could the N2O have damaged or deadened the nerves in that area more longterm? According to Wikipedia, Nitrous Oxide is neurotoxic, but it’s not supposed to be able to cause any serious damage unless the exposure is chronic and sustained:
Indeed, in rodents, short-term exposure results in only mild injury that is rapidly reversed, and permanent neuronal death only occurs after constant and sustained exposure.
-Jevtovic-Todorovic V, Beals J, Benshoff N, Olney JW; Beals; Benshoff; Olney (2003). “Prolonged exposure to inhalational anesthetic nitrous oxide kills neurons in adult rat brain”. Neuroscience.122(3): 609–16. 14622904. doi:10.1016/j.neuroscience.2003.07.012.PMID
My surgery was quick, and would seem to qualify as short term, so I believe the random chance that the N2O had a magnified effect on me and actually deadened my nerve is small.
Perhaps the nitrous oxide simply killed the virus? And maybe pure Oxygen alone would have knocked it off as well? Oxygen can clear out a lot of stuff! Behold, the Hyperbaric Oxygen Chamber! And, although it’s probably inconsequential, as the gas would be absorbed by the oral mucosa anyway, removing my wisdom tooth would have actually allowed for the Nitrous Oxide to penetrate directly to the trouble spot – the part of the gum that once held my wisdom tooth was where I first felt the pain, in both instances.
I realize that N20 has a substantial anti-inflammatory, analgesic, and anxiolytic effect on my nerve(s), that’s the whole point in using it; but that effect is supposed to be short term, and the pain did not return a few hours or even days later when the effects of the N2O would have most surely wore off. In fact, the pain did not return until two years later. Something affected my nerves longterm! This, taken with the effectiveness of my antiviral protocol, leads me to believe that my nerves were keyed up and screaming because of an active viral infection. An infection that the Nitrous Oxide obliterated. I like to think that’s what happened. But I need to do some more research on this beyond Wikipedia!
Another interesting thing, more of a side note –also according to Wikipedia:
Additionally, nitrous oxide depletes Vitamin B12 levels. This can cause serious neurotoxicity with even acute use if the user has preexisting vitamin B12 deficiency. Symptoms of vitamin B12 deficiency, including sensory neuropathy, myelopathy, and encephalopathy can occur within days or weeks of exposure to nitrous oxide anaesthesthia in people with subclinical vitamin B12 deficiency. People with normal vitamin B12 levels have stores to make the effects of nitrous oxide insignificant, unless exposure is repeated and prolonged.
-Giannini, A.J. (1999). Drug Abuse. Los Angeles: Health Information Press.
There’s that B12 again, so very critical for the nervous system.
CFS | Endgame 
Leave a Reply